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  Naoyuki Tsuchiya
   
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œŠ‘®Šw‰ïEŠw‰ï–ðˆõ
  EAmerican College of Rheumatology
EThe Henry Kunkel Society
E“ú–{ƒŠƒEƒ}ƒ`Šw‰ï
E“ú–{“à‰ÈŠw‰ï
E“ú–{–ƉuŠw‰ï
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E“ú–{ƒAƒŒƒ‹ƒM[Šw‰ïi•]‹cˆõj
E“ú–{—Õ°ˆâ“`Œ¤‹†‰ïi•]‹cˆõj
   
œ’˜‘E˜_•¶
  Kono H, et al. Fc ƒÁ RIIB Ile232Thr transmembrane polymorphism associated with human systemic lupus erythematosus decreases affinity to lipid rafts and attenuates inhibitory effects on B cell receptor signaling. Hum Mol Genet 14: 2881-2892, 2005.
  Kuroki K, et al. Extensive polymorphisms of LILRB1 ( ILT2 , LIR1 ) and their association with HLA-DRB1 shared epitope negative rheumatoid arthritis. Hum Mol Genet 14: 2469-2480, 2005.
  Hitomi Y, et al. CD72 polymorphisms associated with alternative splicing modify susceptibility to human systemic lupus erythematosus through epistatic interaction with FCGR2B. Hum Mol Genet 13: 2907-2917, 2004.
  Sakurai D, et al. Crucial role of inhibitor of DNA binding/differentiation in the vascular endothelial growth factor-induced activation and angiogenic processes of human endothelial cells. J Immunol 173: 5801- 5809, 2004.
  Kyogoku C, et al. Association of Fc ƒÁ receptor gene polymorphisms in Japanese patients with systemic lupus erythematosus: Contribution of FCGR2B to the genetic susceptibility to SLE. Arthritis Rheum 46: 1242-1254, 2002.
   
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œtelEfax
  tel : 029-853-3071
  fax : 029-853-3071
   
œƒ[ƒ‹ƒAƒhƒŒƒX
  tsuchiyamd.tsukuba.ac.jp

[—ª—ð]
1983.3 @
“Œ‹ž‘åŠwˆãŠw•”ˆãŠw‰È@‘²‹Æ
1983.6 @
“Œ‹ž‘åŠwˆãŠw•”•‘®•a‰@ˆãˆõiŒ¤Cˆãj
1985.6
“Œ‹ž‘åŠwˆãŠw•”“à‰È•¨——Ö@Šw‹³Žºi•¨—Óà‰Èjˆãˆõ
1987.7
University of New Mexico, Department of Medicine, Research fellow
1988.5
University of Florida , Department of Medicine, Research fellow
1989.12
“Œ‹ž‘åŠwˆãŠw•”“à‰È•¨——Ö@Šw‹³Žºˆãˆõ
1990.8
“Œ‹ž‘åŠwˆãŠw•”“à‰È•¨——Ö@Šw‹³Žº•Žè
1996.6 @
“Œ‹ž‘åŠw‘åŠw‰@ˆãŠwŒnŒ¤‹†‰Èl—Þˆâ“`Šw•ª–앎è
1997.8 @
“Œ‹ž‘åŠw‘åŠw‰@ˆãŠwŒnŒ¤‹†‰Èl—Þˆâ“`Šw•ª–ì•‹³Žö
2006.10
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