| œŽ@–¼ |
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Naoyuki Tsuchiya |
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”ŽŽmiˆãŠwj ( “Œ‹ž‘åŠw ) |
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| œê–啪–ì |
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l—Þˆâ“`ŠwA–Ɖuˆâ“`ŠwAƒŠƒEƒ}ƒ`äPŒ´•a |
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| œŠ‘®Šw‰ïEŠw‰ï–ðˆõ |
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EAmerican College of Rheumatology EThe Henry Kunkel Society E“ú–{ƒŠƒEƒ}ƒ`Šw‰ï E“ú–{“à‰ÈŠw‰ï E“ú–{–ƉuŠw‰ï E“ú–{l—Þˆâ“`Šw‰ï E“ú–{‘gD“K‡«Šw‰ï E“ú–{ƒAƒŒƒ‹ƒM[Šw‰ïi•]‹cˆõj E“ú–{—Õ°ˆâ“`Œ¤‹†‰ïi•]‹cˆõj |
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| œ’˜‘E˜_•¶ |
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Kono H, et al. Fc ƒÁ RIIB Ile232Thr transmembrane polymorphism associated with human systemic lupus erythematosus decreases affinity to lipid rafts and attenuates inhibitory effects on B cell receptor signaling. Hum Mol Genet 14: 2881-2892, 2005. |
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Kuroki K, et al. Extensive polymorphisms of LILRB1 ( ILT2 , LIR1 ) and their association with HLA-DRB1 shared epitope negative rheumatoid arthritis. Hum Mol Genet 14: 2469-2480, 2005. |
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Hitomi Y, et al. CD72 polymorphisms associated with alternative splicing modify susceptibility to human systemic lupus erythematosus through epistatic interaction with FCGR2B. Hum Mol Genet 13: 2907-2917, 2004. |
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Sakurai D, et al. Crucial role of inhibitor of DNA binding/differentiation in the vascular endothelial growth factor-induced activation and angiogenic processes of human endothelial cells. J Immunol 173: 5801- 5809, 2004. |
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Kyogoku C, et al. Association of Fc ƒÁ receptor gene polymorphisms in Japanese patients with systemic lupus erythematosus: Contribution of FCGR2B to the genetic susceptibility to SLE. Arthritis Rheum 46: 1242-1254, 2002. |
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| œŠwpŠÖŒWŽóÜ |
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| œtelEfax |
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tel : 029-853-3071 |
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fax : 029-853-3071 |
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| œƒ[ƒ‹ƒAƒhƒŒƒX |
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tsuchiya md.tsukuba.ac.jp |